Current Opinion in Neurology June 2010 by Peter Goadsby, Wendy Ziai, Renaud Du Pasquier

By Peter Goadsby, Wendy Ziai, Renaud Du Pasquier

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Biological therapies in the spondyloarthritides: the current state. Rheumatology (Oxford) 2004; 43:1072–1084. Copyright © Lippincott Williams & Wilkins. Unauthorized reproduction of this article is prohibited. de Current Opinion in Neurology 2010, 23:234–241 Purpose of review Knowledge of the metabolic and genetic basis of known and previously unknown leukodystrophies is constantly increasing, opening new treatment options such as enzyme replacement or cell-based therapies. This brief review highlights some recent work, particularly emphasizing results from studies in adulthood leukodystrophies.

27 Lanzafame M, Ferrari S, Lattuada E, et al. Mirtazapine in an HIV-1 infected patient with progressive multifocal leukoencephalopathy. Infez Med 2009; 17:35–37. 28 Verma S, Cikurel K, Koralnik IJ, et al. Mirtazapine in progressive multifocal leukoencephalopathy associated with polycythemia vera. J Infect Dis 2007; 196:709–711. 29 Brickelmaier M, Lugovskoy A, Kartikeyan R, et al. Identification and characterization of mefloquine efficacy against JC virus in vitro. Antimicrob Agents Chemother 2009; 53:1840–1849.

Validation of a newborn screening LC–MS/MS method with high sensitivity for practical use in X-ALD. Cell therapies such as bone marrow transplantation have been successfully established in cerebral X-ALD, MLD 15 Deconinck N, Messaaoui A, Ziereisen F, et al. Metachromatic leukodystrophy without arylsulfatase A deficiency: a new case of saposin-B deficiency. Eur J Paediatr Neurol 2008; 12:46–50. 13 Gieselmann V. Metachromatic leukodystrophy: genetics, pathogenesis and  therapeutic options. Acta Paediatr Suppl 2008; 97:15–21.

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