Concise Neurology by Alberto J. Espay MD MSc, José Biller MD FACP FAAN FAHA

By Alberto J. Espay MD MSc, José Biller MD FACP FAAN FAHA

Concise Neurology offers neurology content material in a logical series inside of every one of ten chapters. almost each subject of curiosity to clinicians is roofed in a single or pages to facilitate effective evaluate of the middle options in analysis and therapy for every syndrome or affliction. the writer has maintained a thematic team spirit throughout comparable pages, which permits for random examining to be virtually as fruitful as sequential interpreting. for instance, a reader who opens the "anticoagulation" web page will discover a capsulized evaluation of the heparin, warfarin, and thrombolysis stroke trials and the facts aiding the present healing directions. Flipping via neighboring pages will speedily refresh the location of the clinically suitable thrombophilias in terms of the coagulation pathway, symptoms for lifelong anticoagulation, factors of hyperhomocysteinemia, and the naked bones of an important etiologies of strokes within the young.
 
The booklet offers larger point content material than the scholar "In a web page" textual content, covers each one subject in additional intensity, and addresses concerns with regards to pathophysiology and neuropathology of ailments. Concise Neurology could be a welcome source for busy practitioners, citizens, fellows, scientific scholars, and allied health and wellbeing care execs within the neurosciences who require a speedy, readable assessment of a posh subject.

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Metachromatic Leukodystrophy (Arylsulfatase A or Saposin B Deficiency) Deficiency of arylsulfatase A or its activator, saposin B, leads to accumulation of cerebroside sulfate, which causes progressive (frontal-predominant) central and peripheral demyelination. The NCV are <30 m/s even when reflexes are brisk. Progressive spastic quadripar esis may be associated with blindness, seizur es, dementia, and peripheral neur opathy. Increased urinary sulfatides with normal arylsulfatase A levels are seen in MLD due to saposin B deficiency.

Types A and B appear most often in Jewish families. Type C affects all ethnic groups and is the most common. Ataxia and dystonia are followed by supranuclear vertical gaze palsy, seizures, and dementia. Hepatosplenomegaly often coexists. Foamy (lipid-laden) cells or “sea-blue histiocytes” in the liver and bone marrow are diagnostic. Metachromatic Leukodystrophy (Arylsulfatase A or Saposin B Deficiency) Deficiency of arylsulfatase A or its activator, saposin B, leads to accumulation of cerebroside sulfate, which causes progressive (frontal-predominant) central and peripheral demyelination.

ADHD affects about 5% of school-aged children worldwide, predominantly males (3:1 to 8:1). About one-third of ADHD cases have at least one ADHD parent. The risk to first-degree relatives of affected individual is 8-10 times that of the general population. ADHD persists into adolescence in about 30%–50% of affected individuals. The DSM-IV-TR distinguishes two dimensions in the behavior of individuals with ADHD: inattention and hyperactivity/impulsivity. Based on these two dimensions, thr ee behavioral subtypes ar e identif ed: • Primarily inattentive • Primarily hyperactive/impulsive • Combined inattentive and hyperactive/impulsive For a diagnosis of ADHD , the patient must present with six or more symptoms of inattention, six or more symptoms of hyperactivity/impulsivity, or both for an inattentive, hyperactive/impulsive, or combined subtype diagnosis.

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